Monday, March 30, 2009
Sunday, March 29, 2009
The amazing human brain
I saw the movie a beautiful mind couple of years ago
and i was shockedthe human brain which is still the worlds biggest mystery...
Can behave like that
Schizophrenia iwas always scared of it
I mean the brain can be so cmplicated yet so scary i never thought
but A beautiful mind has change things for me
Idnt belive putting up a war against schizophrenia but accepting it...
iknow a freind who has schizophrenia
i was scared when found it
i have hope that such people can live a normal life
Its scary to have halucinations and here voices
in marathi they are called "manache khel"
thats why people see and belive ghosts
my mm belives training the mind s she always made me byheart manache sloke and pray to lord hanuman
my teacher says mind iis awild thing like you saddle a horse you t saddle it should always be your slave
in bhagwat geeta also it is said the brain should have ful;ll over the mind
then again some thing goes withat beautiful mind
luck fate...
GOD
ALL OTHER ILLNESSES ARE BETTER THAN PSYCATRIC ILLNESSSES
people say does heart keeps a man alive or the brain...
HONESTLY ITS THE BRAIN
AND ONLY FAITH IIN GOD KEEPS IT ALIVE AND IN GOOD HEALTH...
I DONT WHATS GOING TO HAPPENT NEXT MINUITE BUT THIS MINUITE I BELIVE AND HOPE THAT AM BLESSED WITH GOOD HEALTH PHYSICALLY AND PSYCOLOGICALLY...AND EVERYBODY ELSE TOO
AND I LOVE THE MOVIE BEAUTIFUL MIND
iTS FULL OF HOPE AND POSITIVE ENERGY IT MAKES FEEL GOD IS CLOSE TO ME.. I SAW IT COUPLE OF YEARS AGO.
if possible it better to accept schizophrenia it helps to live a normal life...i belive that and keep praying.
tHIGS TO LIVE TRYING TO RUN AWAY FROM IT OR ACCEPT AND LIVE WITH IT
I AGREE WITH IT ITS NOT SCXARY ONCE YOU NOW ITS HAPPEENING BECAUSE UR ILL... AND ITS NOT REAL AND WONT GO AWAY BECAUSE ITS INCURABLE IT MAKES LIFE EASY WONDER IF ANY BODY AGREES WITH ME.
About schizophrenia:
(pronounced /ˌskɪtsəˈfrɛniə/ or /ˌskɪtsəˈfriːniə/), from the Greek roots schizein (σχίζειν, "to split") and phrēn, phren- (φρήν, φρεν-, "mind") is a psychiatric diagnosis that describes a mental disorder characterized by abnormalities in the perception or expression of reality. It most commonly manifests as auditory hallucinations, paranoid or bizarre delusions, or disorganized speech and thinking with significant social or occupational dysfunction. Onset of symptoms typically occurs in young adulthood,[1] with approximately 0.4–0.6%[2][3] of the population affected. Diagnosis is based on the patient's self-reported experiences and observed behavior. No laboratory test for schizophrenia currently exists.[4]
Studies suggest that genetics, early environment, neurobiology, psychological and social processes are important contributory factors; some recreational and prescription drugs appear to cause or worsen symptoms. Current psychiatric research is focused on the role of neurobiology, but no single organic cause has been found. Due to the many possible combinations of symptoms, there is debate about whether the diagnosis represents a single disorder or a number of discrete syndromes. For this reason, Eugen Bleuler termed the disease the schizophrenias (plural) when he coined the name. Despite its etymology, schizophrenia is not the same as dissociative identity disorder, previously known as multiple personality disorder or split personality. Schizophrenia has been commonly and erroneously confused with multiple personality order.[5]
Increased dopamine activity in the mesolimbic pathway of the brain is consistently found in schizophrenic individuals. The mainstay of treatment is antipsychotic medication; this type of drug primarily works by suppressing dopamine activity. Dosages of antipsychotics are generally lower than in the early decades of their use. Psychotherapy, and vocational and social rehabilitation are also important. In more serious cases—where there is risk to self and others—involuntary hospitalization may be necessary, although hospital stays are less frequent and for shorter periods than they were in previous years.[6]
The disorder is thought to mainly affect cognition, but it also usually contributes to chronic problems with behavior and emotion. People with schizophrenia are likely to have additional (comorbid) conditions, including major depression and anxiety disorders;[7] the lifetime occurrence of substance abuse is around 40%. Social problems, such as long-term unemployment, poverty and homelessness, are common. Furthermore, the average life expectancy of people with the disorder is 10 to 12 years less than those without, due to increased physical health problems and a higher suicide rate.[8]
Causes:
While the reliability of the diagnosis introduces difficulties in measuring the relative effect of genes and environment (for example, symptoms overlap to some extent with severe bipolar disorder or major depression), evidence suggests that genetic and environmental factors can act in combination to result in schizophrenia.[48] Evidence suggests that the diagnosis of schizophrenia has a significant heritable component but that onset is significantly influenced by environmental factors or stressors.[49] The idea of an inherent vulnerability (or diathesis) in some people, which can be unmasked by biological, psychological or environmental stressors, is known as the stress-diathesis model.[50] The idea that biological, psychological and social factors are all important is known as the "biopsychosocial" model.
Genetic
Estimates of the heritability of schizophrenia tend to vary owing to the difficulty of separating the effects of genetics and the environment although twin studies have suggested a high level of heritability.[51] It has been suggested that schizophrenia is a condition of complex inheritance, with several genes possibly interacting to generate risk for schizophrenia or the separate components that can co-occur leading to a diagnosis.[52] These genes appear to be non-specific, in that they may raise the risk of developing other psychiatric disorders such as bipolar disorder.[53] However, recent metaanalyses of linkage studies have produced conflicting findings.[54] Larger-scale, thus more sensitive genome-wide association studies are being conducted.[55] Schizophrenia has also been associated with rare deletions or duplications of tiny DNA sequences (known as copy number variants) disproportionately occurring within genes involved in neuronal signaling and brain development.[56][57]
There is little doubt about the existence of a fecundity deficit in schizophrenia. Affected individuals have fewer children than the population as a whole. This reduction is of the order of 70% in males and 30% in females. The central genetic paradox of schizophrenia is why if the disease is associated with a biological disadvantage is this variation not selected out? To balance such a significant disadvantage, a substantial and universal advantage must be exist. Insofar, all theories of a putative advantage were disproved or remain unsubstantiated.[58][59]
Prenatal
Causal factors are thought to initially come together in early neurodevelopment to increase the risk of later developing schizophrenia. One curious finding is that people diagnosed with schizophrenia are more likely to have been born in winter or spring, (at least in the northern hemisphere).[60] There is now evidence that prenatal exposure to infections increases the risk for developing schizophrenia later in life, providing additional evidence for a link between in utero developmental pathology and risk of developing the condition.[61]
Social
Living in an urban environment has been consistently found to be a risk factor for schizophrenia.[62][45] Social disadvantage has been found to be a risk factor, including poverty[63] and migration related to social adversity, racial discrimination, family dysfunction, unemployment or poor housing conditions.[64] Childhood experiences of abuse or trauma have also been implicated as risk factors for a diagnosis of schizophrenia later in life.[65][66] Parenting is not held responsible for schizophrenia but unsupportive dysfunctional relationships may contribute to an increased risk.[67][68]
Drugs
See also: Dual diagnosis
Although about half of all patients with schizophrenia abuse drugs or alcohol, a clear causal connection between drug use and schizophrenia has been difficult to prove. The two most often used explanations for this are "substance use causes schizophrenia" and "substance use is a consequence of schizophrenia", and they both may be correct.[69] A 2007 meta-analysis estimated that cannabis use is statistically associated with a dose-dependent increase in risk of development of psychotic disorders, including schizophrenia.[70] There is little evidence to suggest that other drugs such as alcohol cause psychosis, or that psychotic individuals choose specific drugs to self-medicate; there is some support for the theory that they use drugs to cope with unpleasant states such as depression, anxiety, boredom and loneliness
SOURCE AND MORE INFO:
http://en.wikipedia.org/wiki/Schizophrenia
tHE STORU OF A BEAUTIFUL MIND DO WATCH THE MOVIE:
John Nash (Russell Crowe) arrives at Princeton University as a new graduate student. He is a recipient of the prestigious Carnegie Prize for mathematics; although he was promised a single room, his roommate Charles (Paul Bettany), a literature student, greets him as he moves in and soon becomes his best friend. Nash also meets a group of other promising math and science graduate students, Martin Hansen (Josh Lucas), Sol (Adam Goldberg), Ainsley, and Bender (Anthony Rapp), with whom he strikes up an awkward friendship. Nash admits to Charles that he is better with numbers than people, which comes as no surprise to them after watching his largely unsuccessful attempts at conversation with the women at the local bar.
The headmaster of Princeton informs Nash, who has missed many of his classes, that he cannot begin work until he finishes a thesis paper, prompting him to seek a truly original idea for the paper. A woman at the bar is what ultimately inspires his fruitful work in the concept of governing dynamics, a theory in mathematical economics.
After the conclusion of Nash's studies as a student at Princeton, he accepts a prestigious appointment at the Massachusetts Institute of Technology (MIT), along with his friends Sol and Bender.
Five years later, while teaching a class on calculus at MIT, he places a particularly interesting problem on the chalkboard that he dares his students to solve. When his student Alicia Larde (Jennifer Connelly) comes to his office to discuss the problem, the two fall in love and eventually marry.
On a return visit to Princeton, Nash runs into his former roommate Charles and meets Charles' young niece Marcee (Vivien Cardone), whom he adores. Nash is invited to a secret Department of Defense facility in the Pentagon to crack a complex encryption of an enemy telecommunication. Nash is able to decipher the code mentally, to the astonishment of other codebreakers. Here, he encounters the mysterious William Parcher (Ed Harris), who belongs to the United States Department of Defense.
Parcher observes Nash's performance from above, while partially concealed behind a screen. Parcher gives Nash a new assignment to look for patterns in magazines and newspapers, ostensibly to thwart a Soviet plot. He must write a report of his findings and place them in a specified mailbox. After being chased by Russian agents and an exchange of gunfire, Nash becomes increasingly paranoid and begins to behave erratically.
After observing this erratic behavior, Alicia informs a psychiatric hospital. Later, while delivering a guest lecture at Princeton University, Nash realizes that he is being watched by a hostile group of people; although, he attempts to flee, he is forcibly sedated and sent to a psychiatric facility. Nash's internment seemingly confirms his belief that the Soviets are trying to extract information from him. He views the officials of the psychiatric facility as Soviet kidnappers. At one point, he insanely tries to dig out of his arm an implant he received at The Pentagon, causing much bleeding.
Alicia, desperate to help her husband, visits the mailbox and retrieves the never-opened "top secret" documents that Nash had delivered there. When confronted with this evidence, Nash is finally convinced that he has been hallucinating. The Department of Defense agent William Parcher and Nash's secret assignment to decode Soviet messages was in fact all a delusion. Even more surprisingly, Nash's friend Charles and his niece Marcee are also only products of Nash's mind.
After a series of insulin shock therapy sessions, Nash is released on the condition that he agrees to take antipsychotic medication; however, the drugs create negative side-effects that affect his sexual and emotional relationship with his wife and, most dramatically, his intellectual capacity. Frustrated, Nash secretly stops taking his medication and hoards his pills, triggering a relapse of his psychosis.
While bathing his infant son, Nash becomes distracted and wanders off. Alicia is hanging laundry in the backyard and observes that the back gate is open. She discovers that Nash has turned an abandoned shed in a nearby grove of trees into an office for his work for Parcher. Upon realizing what has happened, Alicia runs into the house to confront Nash and barely saves their child from drowning in the bathtub. When she confronts him, Nash claims that his friend Charles was watching their son. Alicia runs to the phone to call the psychiatric hospital for emergency assistance. Nash suddenly sees Parcher who urges him to kill his wife, but Nash angrily refuses to do such a thing. After Parcher points a gun at her, Nash lunges for him, accidentally knocking Alicia to the ground. Alicia flees the house in fear with their child, but Nash steps in front of her car to prevent her from leaving. After a moment, Nash realizes that Marcee is a hallucination, because although years have passed since their first encounter, Marcee has remained exactly the same age and is still a little girl. Realizing the implications of this fact, he tells Alicia, "She never gets old." Only then does he accept that although all three people seem completely real, they are in fact part of his hallucinations.
Caught between the intellectual paralysis of the antipsychotic drugs and his delusions, Nash and Alicia decide to try to live with his abnormal condition. Nash consciously says goodbye to the three of them forever in his attempts to ignore his hallucinations and not feed "his demons"; however, he thanks Charles for being his best friend over the years, and says a tearful goodbye to Marcee, stroking her hair and calling her "baby girl", telling them both he would not speak to them anymore. They still continue to haunt him, with Charles mocking him for cutting off their friendship, but Nash learns to ignore them.
Nash grows older and approaches his old friend and intellectual rival Martin Hansen, now head of the Princeton mathematics department, who grants him permission to work out of the library and audit classes. Even though Nash still suffers from hallucinations and mentions taking newer medications, he is ultimately able to live with and largely ignore his psychotic episodes. He takes his situation in stride and humorously checks to ensure that any new acquaintances are in fact real people, not hallucinations.
Nash eventually earns the privilege of teaching again. He is honored by his fellow professors for his achievement in mathematics, and goes on to win the Nobel Memorial Prize in Economics for his revolutionary work on game theory. Nash and Alicia are about to leave the auditorium in Stockholm, when Nash sees Charles, Marcee and Parcher standing and watching him with blank expressions on their faces. Alicia asks Nash, "What's wrong?" Nash replies, "Nothing. Nothing at all." With that, they both leave the auditorium.
and i was shockedthe human brain which is still the worlds biggest mystery...
Can behave like that
Schizophrenia iwas always scared of it
I mean the brain can be so cmplicated yet so scary i never thought
but A beautiful mind has change things for me
Idnt belive putting up a war against schizophrenia but accepting it...
iknow a freind who has schizophrenia
i was scared when found it
i have hope that such people can live a normal life
Its scary to have halucinations and here voices
in marathi they are called "manache khel"
thats why people see and belive ghosts
my mm belives training the mind s she always made me byheart manache sloke and pray to lord hanuman
my teacher says mind iis awild thing like you saddle a horse you t saddle it should always be your slave
in bhagwat geeta also it is said the brain should have ful;ll over the mind
then again some thing goes withat beautiful mind
luck fate...
GOD
ALL OTHER ILLNESSES ARE BETTER THAN PSYCATRIC ILLNESSSES
people say does heart keeps a man alive or the brain...
HONESTLY ITS THE BRAIN
AND ONLY FAITH IIN GOD KEEPS IT ALIVE AND IN GOOD HEALTH...
I DONT WHATS GOING TO HAPPENT NEXT MINUITE BUT THIS MINUITE I BELIVE AND HOPE THAT AM BLESSED WITH GOOD HEALTH PHYSICALLY AND PSYCOLOGICALLY...AND EVERYBODY ELSE TOO
AND I LOVE THE MOVIE BEAUTIFUL MIND
iTS FULL OF HOPE AND POSITIVE ENERGY IT MAKES FEEL GOD IS CLOSE TO ME.. I SAW IT COUPLE OF YEARS AGO.
if possible it better to accept schizophrenia it helps to live a normal life...i belive that and keep praying.
tHIGS TO LIVE TRYING TO RUN AWAY FROM IT OR ACCEPT AND LIVE WITH IT
I AGREE WITH IT ITS NOT SCXARY ONCE YOU NOW ITS HAPPEENING BECAUSE UR ILL... AND ITS NOT REAL AND WONT GO AWAY BECAUSE ITS INCURABLE IT MAKES LIFE EASY WONDER IF ANY BODY AGREES WITH ME.
About schizophrenia:
(pronounced /ˌskɪtsəˈfrɛniə/ or /ˌskɪtsəˈfriːniə/), from the Greek roots schizein (σχίζειν, "to split") and phrēn, phren- (φρήν, φρεν-, "mind") is a psychiatric diagnosis that describes a mental disorder characterized by abnormalities in the perception or expression of reality. It most commonly manifests as auditory hallucinations, paranoid or bizarre delusions, or disorganized speech and thinking with significant social or occupational dysfunction. Onset of symptoms typically occurs in young adulthood,[1] with approximately 0.4–0.6%[2][3] of the population affected. Diagnosis is based on the patient's self-reported experiences and observed behavior. No laboratory test for schizophrenia currently exists.[4]
Studies suggest that genetics, early environment, neurobiology, psychological and social processes are important contributory factors; some recreational and prescription drugs appear to cause or worsen symptoms. Current psychiatric research is focused on the role of neurobiology, but no single organic cause has been found. Due to the many possible combinations of symptoms, there is debate about whether the diagnosis represents a single disorder or a number of discrete syndromes. For this reason, Eugen Bleuler termed the disease the schizophrenias (plural) when he coined the name. Despite its etymology, schizophrenia is not the same as dissociative identity disorder, previously known as multiple personality disorder or split personality. Schizophrenia has been commonly and erroneously confused with multiple personality order.[5]
Increased dopamine activity in the mesolimbic pathway of the brain is consistently found in schizophrenic individuals. The mainstay of treatment is antipsychotic medication; this type of drug primarily works by suppressing dopamine activity. Dosages of antipsychotics are generally lower than in the early decades of their use. Psychotherapy, and vocational and social rehabilitation are also important. In more serious cases—where there is risk to self and others—involuntary hospitalization may be necessary, although hospital stays are less frequent and for shorter periods than they were in previous years.[6]
The disorder is thought to mainly affect cognition, but it also usually contributes to chronic problems with behavior and emotion. People with schizophrenia are likely to have additional (comorbid) conditions, including major depression and anxiety disorders;[7] the lifetime occurrence of substance abuse is around 40%. Social problems, such as long-term unemployment, poverty and homelessness, are common. Furthermore, the average life expectancy of people with the disorder is 10 to 12 years less than those without, due to increased physical health problems and a higher suicide rate.[8]
Causes:
While the reliability of the diagnosis introduces difficulties in measuring the relative effect of genes and environment (for example, symptoms overlap to some extent with severe bipolar disorder or major depression), evidence suggests that genetic and environmental factors can act in combination to result in schizophrenia.[48] Evidence suggests that the diagnosis of schizophrenia has a significant heritable component but that onset is significantly influenced by environmental factors or stressors.[49] The idea of an inherent vulnerability (or diathesis) in some people, which can be unmasked by biological, psychological or environmental stressors, is known as the stress-diathesis model.[50] The idea that biological, psychological and social factors are all important is known as the "biopsychosocial" model.
Genetic
Estimates of the heritability of schizophrenia tend to vary owing to the difficulty of separating the effects of genetics and the environment although twin studies have suggested a high level of heritability.[51] It has been suggested that schizophrenia is a condition of complex inheritance, with several genes possibly interacting to generate risk for schizophrenia or the separate components that can co-occur leading to a diagnosis.[52] These genes appear to be non-specific, in that they may raise the risk of developing other psychiatric disorders such as bipolar disorder.[53] However, recent metaanalyses of linkage studies have produced conflicting findings.[54] Larger-scale, thus more sensitive genome-wide association studies are being conducted.[55] Schizophrenia has also been associated with rare deletions or duplications of tiny DNA sequences (known as copy number variants) disproportionately occurring within genes involved in neuronal signaling and brain development.[56][57]
There is little doubt about the existence of a fecundity deficit in schizophrenia. Affected individuals have fewer children than the population as a whole. This reduction is of the order of 70% in males and 30% in females. The central genetic paradox of schizophrenia is why if the disease is associated with a biological disadvantage is this variation not selected out? To balance such a significant disadvantage, a substantial and universal advantage must be exist. Insofar, all theories of a putative advantage were disproved or remain unsubstantiated.[58][59]
Prenatal
Causal factors are thought to initially come together in early neurodevelopment to increase the risk of later developing schizophrenia. One curious finding is that people diagnosed with schizophrenia are more likely to have been born in winter or spring, (at least in the northern hemisphere).[60] There is now evidence that prenatal exposure to infections increases the risk for developing schizophrenia later in life, providing additional evidence for a link between in utero developmental pathology and risk of developing the condition.[61]
Social
Living in an urban environment has been consistently found to be a risk factor for schizophrenia.[62][45] Social disadvantage has been found to be a risk factor, including poverty[63] and migration related to social adversity, racial discrimination, family dysfunction, unemployment or poor housing conditions.[64] Childhood experiences of abuse or trauma have also been implicated as risk factors for a diagnosis of schizophrenia later in life.[65][66] Parenting is not held responsible for schizophrenia but unsupportive dysfunctional relationships may contribute to an increased risk.[67][68]
Drugs
See also: Dual diagnosis
Although about half of all patients with schizophrenia abuse drugs or alcohol, a clear causal connection between drug use and schizophrenia has been difficult to prove. The two most often used explanations for this are "substance use causes schizophrenia" and "substance use is a consequence of schizophrenia", and they both may be correct.[69] A 2007 meta-analysis estimated that cannabis use is statistically associated with a dose-dependent increase in risk of development of psychotic disorders, including schizophrenia.[70] There is little evidence to suggest that other drugs such as alcohol cause psychosis, or that psychotic individuals choose specific drugs to self-medicate; there is some support for the theory that they use drugs to cope with unpleasant states such as depression, anxiety, boredom and loneliness
SOURCE AND MORE INFO:
http://en.wikipedia.org/wiki/Schizophrenia
tHE STORU OF A BEAUTIFUL MIND DO WATCH THE MOVIE:
John Nash (Russell Crowe) arrives at Princeton University as a new graduate student. He is a recipient of the prestigious Carnegie Prize for mathematics; although he was promised a single room, his roommate Charles (Paul Bettany), a literature student, greets him as he moves in and soon becomes his best friend. Nash also meets a group of other promising math and science graduate students, Martin Hansen (Josh Lucas), Sol (Adam Goldberg), Ainsley, and Bender (Anthony Rapp), with whom he strikes up an awkward friendship. Nash admits to Charles that he is better with numbers than people, which comes as no surprise to them after watching his largely unsuccessful attempts at conversation with the women at the local bar.
The headmaster of Princeton informs Nash, who has missed many of his classes, that he cannot begin work until he finishes a thesis paper, prompting him to seek a truly original idea for the paper. A woman at the bar is what ultimately inspires his fruitful work in the concept of governing dynamics, a theory in mathematical economics.
After the conclusion of Nash's studies as a student at Princeton, he accepts a prestigious appointment at the Massachusetts Institute of Technology (MIT), along with his friends Sol and Bender.
Five years later, while teaching a class on calculus at MIT, he places a particularly interesting problem on the chalkboard that he dares his students to solve. When his student Alicia Larde (Jennifer Connelly) comes to his office to discuss the problem, the two fall in love and eventually marry.
On a return visit to Princeton, Nash runs into his former roommate Charles and meets Charles' young niece Marcee (Vivien Cardone), whom he adores. Nash is invited to a secret Department of Defense facility in the Pentagon to crack a complex encryption of an enemy telecommunication. Nash is able to decipher the code mentally, to the astonishment of other codebreakers. Here, he encounters the mysterious William Parcher (Ed Harris), who belongs to the United States Department of Defense.
Parcher observes Nash's performance from above, while partially concealed behind a screen. Parcher gives Nash a new assignment to look for patterns in magazines and newspapers, ostensibly to thwart a Soviet plot. He must write a report of his findings and place them in a specified mailbox. After being chased by Russian agents and an exchange of gunfire, Nash becomes increasingly paranoid and begins to behave erratically.
After observing this erratic behavior, Alicia informs a psychiatric hospital. Later, while delivering a guest lecture at Princeton University, Nash realizes that he is being watched by a hostile group of people; although, he attempts to flee, he is forcibly sedated and sent to a psychiatric facility. Nash's internment seemingly confirms his belief that the Soviets are trying to extract information from him. He views the officials of the psychiatric facility as Soviet kidnappers. At one point, he insanely tries to dig out of his arm an implant he received at The Pentagon, causing much bleeding.
Alicia, desperate to help her husband, visits the mailbox and retrieves the never-opened "top secret" documents that Nash had delivered there. When confronted with this evidence, Nash is finally convinced that he has been hallucinating. The Department of Defense agent William Parcher and Nash's secret assignment to decode Soviet messages was in fact all a delusion. Even more surprisingly, Nash's friend Charles and his niece Marcee are also only products of Nash's mind.
After a series of insulin shock therapy sessions, Nash is released on the condition that he agrees to take antipsychotic medication; however, the drugs create negative side-effects that affect his sexual and emotional relationship with his wife and, most dramatically, his intellectual capacity. Frustrated, Nash secretly stops taking his medication and hoards his pills, triggering a relapse of his psychosis.
While bathing his infant son, Nash becomes distracted and wanders off. Alicia is hanging laundry in the backyard and observes that the back gate is open. She discovers that Nash has turned an abandoned shed in a nearby grove of trees into an office for his work for Parcher. Upon realizing what has happened, Alicia runs into the house to confront Nash and barely saves their child from drowning in the bathtub. When she confronts him, Nash claims that his friend Charles was watching their son. Alicia runs to the phone to call the psychiatric hospital for emergency assistance. Nash suddenly sees Parcher who urges him to kill his wife, but Nash angrily refuses to do such a thing. After Parcher points a gun at her, Nash lunges for him, accidentally knocking Alicia to the ground. Alicia flees the house in fear with their child, but Nash steps in front of her car to prevent her from leaving. After a moment, Nash realizes that Marcee is a hallucination, because although years have passed since their first encounter, Marcee has remained exactly the same age and is still a little girl. Realizing the implications of this fact, he tells Alicia, "She never gets old." Only then does he accept that although all three people seem completely real, they are in fact part of his hallucinations.
Caught between the intellectual paralysis of the antipsychotic drugs and his delusions, Nash and Alicia decide to try to live with his abnormal condition. Nash consciously says goodbye to the three of them forever in his attempts to ignore his hallucinations and not feed "his demons"; however, he thanks Charles for being his best friend over the years, and says a tearful goodbye to Marcee, stroking her hair and calling her "baby girl", telling them both he would not speak to them anymore. They still continue to haunt him, with Charles mocking him for cutting off their friendship, but Nash learns to ignore them.
Nash grows older and approaches his old friend and intellectual rival Martin Hansen, now head of the Princeton mathematics department, who grants him permission to work out of the library and audit classes. Even though Nash still suffers from hallucinations and mentions taking newer medications, he is ultimately able to live with and largely ignore his psychotic episodes. He takes his situation in stride and humorously checks to ensure that any new acquaintances are in fact real people, not hallucinations.
Nash eventually earns the privilege of teaching again. He is honored by his fellow professors for his achievement in mathematics, and goes on to win the Nobel Memorial Prize in Economics for his revolutionary work on game theory. Nash and Alicia are about to leave the auditorium in Stockholm, when Nash sees Charles, Marcee and Parcher standing and watching him with blank expressions on their faces. Alicia asks Nash, "What's wrong?" Nash replies, "Nothing. Nothing at all." With that, they both leave the auditorium.
Subscribe to:
Posts (Atom)
